医学专业论文参考文献

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医学专业论文参考文献

　　从小学、初中、高中到大学乃至工作，大家都尝试过写论文吧，论文是指进行各个学术领域的研究和描述学术研究成果的文章。那么你知道一篇好的论文该怎么写吗？以下是小编收集整理的医学专业论文参考文献，希望对大家有所帮助。

医学专业论文参考文献

　　医学论文参考文献一：

　　[1]高松柏.国际婴幼儿食品研究新动态[J].中国乳品工业，2007(4)：36-40.

　　[2]赵学伟.食品挤压膨化机理研宄进展[J].粮食加工，2010,35 (2) :59-65.

　　[3]丁小林.大豆食品的营养价值与功能特性[J].食品研宄与开发，2004,25,(6)：101.

　　[4]韩立德，盖钧镒，张文明.大豆营养成分研究现状[J].种子，2003(5)： 58.

　　[5]夏剑秋，张毅方.大豆中主要营养成分和微量元素的功能作用[J].中国油脂，2007,3(1)：72.

　　[6]姜中航，婴儿配方奶粉生产中部分应用大豆分离蛋白的研宂[J].大豆科技，2009(2)：41-43.

　　[7]高连胜，李铁红，崔洪滨，等.适合婴儿配方奶粉用蛋白的研宄[J].食品工业，2009(4)：52.

　　[8]张瑞岩.豆基婴儿配方粉的研究[D].东北农业大学，2009.

　　[9]张连慧.大豆分离蛋白在乳及乳制品中的应用[J].食品安全导刊，2009(5)： 46.

　　[10]陈莉.无糖速溶速凝豆奶粉的研制[D].江南大学，2005.

　　[11]朱彦群，倪群.牛奶大豆双蛋白营养酸奶的研究[J].食品研究与开发，2007,28(4) :110-111.

　　[12]高蝉，高连胜.大豆分离蛋白在婴儿配方粉中的应用研宄[J].中国乳业.2008(3)56-58.

　　[13]汪之顼.叶黄素和玉米黄质对视觉发育和健康的影响[J].国外医学卫生学分册.2008(3)： 154-159.

　　[14]李忠海，李安平.婴幼儿营养米粉配方的优化设计[J].中南林学院学报，2001,21(1)：54-57.

　　[15]AnastaseH. Evaluation of rice flour modified by extrusion cooking [J]. Journal ofcereal Science, 2006(43) :38-46.

　　[16]MercedsB,ConsueloM,PerezO, etal. Varietal differences in properties of extrusioncooked rice flour[J]. Food Chemistry, 1986, 19(3)： 173-187.

　　医学论文参考文献二：

　　[1]陆红梅.视疲劳中医理论探讨及用药规律分析[D];扬州大学，2010.

　　[2]叶建东.视疲劳诊断、病因及其治疗的研究进展[J].中国疗养医学，2014,03)： 210-2.

　　[3]张仁.透刺为主治疗视疲劳的临床观察[J].上海针灸杂志，2007,09)： 9-11.

　　[4]李尖.视屏显示终端与视疲劳及潘光不正[J].临床医药实践，2006,08)：589-91.

　　[5]张秀云.新型饮食疗法对2型糖尿病患者的干预效果[J].中华护理杂志，2004, 10)： 22-4.

　　[6]张广琴.中青年视疲劳与视屏终端[J].大家健康(学术版)，2011,19)：16-7.

　　[7]陈丽君.利用传统医学经络调理法改善视疲劳的临床观察[D];北京中医药大学，2011.

　　[8]潘小云.加味六君子汤治疗视疲劳40例疗效观察[J].浙江中医杂志，2010,02)： 130.

　　[9]黄文刚.中药熏蒸联合眼部穴位按摩改善视疲劳症状的疗效观察[J].中国中医眼科杂志，2014,04)： 251-4.

　　[10]Johnson E J. A biological role of lutein [J]. Food Rev Int,2004,20(1); 1-16.

　　[11]Christen W Q Liu S,Glynn R J,et al. Dietary carotenoids, vitamins C and E, and risk of cataract in women [J]. Arch Ophthalmol, 2008, 126(1)： 102-9.

　　[12]Bone R A,Landrum J T,Dixon Z,et al. Lutein and zeaxanthin in the eyes,serum and diet of human subjects [J]. Exp Eye Res, 2000,71(3)： 239-45.

　　医学论文参考文献三：

　　[1]程牛亮.生物化学[M].北京：高等教育出版社，2011:299-311.]

　　[2]毛羽扬.食盐在烹饪调味中的作用[J].中国调味品，1999,5.

　　[3]岩濑荣一.盐及其化学[M].北京：轻工业出版社，1960:10-36.

　　[4]苏敬.新修本草[M].群联出版社，1955.

　　[5]金明宫.竹盐的烤熔熏制法[J].中小企业科技，2003,7:019.

　　[6]潘进富，张成霞.化学教育[M].1999,20 (6)：3-4.

　　[7]李卫平.功能性保健食盐的开发研究[J].海湖盐科技资料，2000(8)：1-6.

　　[8]王琦.王琦方药应用31论[M].2012:250-267.

　　[9]陈敏章，邵丙杨.中华内科学[M],北京：人民卫生出版社，2006:2477-2481.

　　[10]高钦颖.韩国仁山竹盐治疗胃病15例[J].陕西中医，1994,15(7)：298.

　　[11]美国NKF-K/DOQI工作组.慢性肾脏病及透析的临床实践指南[M].北京：人民卫生出版社，2003(1).

　　[12]郭义敏，刘颖，陈历明.近端肾小管碳酸氢根重吸收的分子机制及代谢性酸中毒[J].生理学报，2014,66(4)： 398~414.

　　[13]Moon, et al. The Intrinsic Antimicrobial Activity of Bamboo Salt against Salmonella enteritidis[J]. Molecular and Cellular Toxicology, 2010,6(1)： 109-109.

　　医学论文参考文献

　　[1]Weaver A., da Silva A. G, Nuttall R. K.，et al. An elevated matrix metalloproteinase(MMP) in an animal model of multiple sclerosis is protective by affecting Thl/Th2polarization. The FASEB journal. 2005. 19(12): p. 1668-1670.

　　[2]Venteo L_，Bourlet T” Renois F” et al. Enterovirus-related activation of thecardiomyocyte mitochondrial apoptotic pathway in patients with acute myocarditis.European heart journal. 2010. 31(6): p. 728-736.

　　[3]Rose N. R. The adjuvant effect in infection and autoimmunity. Clinical reviews inallergy & immunology. 2008. 34(3): p. 279-282.

　　[4]陈瑞珍，解玉泉.病毒性心肌炎向扩张型心肌病演变的免疫学机制.诊断学理论与实践-2012. 10(5): p. 414-417.

　　[5]Camporeale A., Marino F” Papageorgiou A.，et al. STAT3 activity is necessary andsufficient for the development of immune “ mediated myocarditis in mice andpromotes progression to dilated cardiomyopathy. EMBO molecular medicine. 2013.5(4): p. 572-590.

　　[6]Zhu J.,Paul W. E. CD4 T cells: fates, functions, and faults. Blood. 2008. 112(5): p.1557-1569.

　　[7]Korn T.，Bettelli E?，Oukka M.，et al. IL-17 and Thl7 Cells. Annual review ofimmunology. 2009. 27: p. 485-517.

　　[8]Yu Y” Liu Y-，Shi F.-D., et al. Cutting edge: leptin-induced RORyt expression in CD4+T cells promotes Thl7 responses in systemic lupus erythematosus. The Journal ofImmunology. 2013. 190(7): p. 3054-3058.

　　[9]van Bruggen N.,Ouyang W. Thl7 Cells at the Crossroads of Autoimmunity,Inflammation, and Atherosclerosis. Immunity. 2014. 40(1): p. 10-12.

　　[10]Annnn2:iatoF., Cosmi L., Santarlasci V.，et al. Phenotypic and functional features ofhuman Thl7 cells. The Journal of experimental medicine. 2007. 204(8): p. 1849-1861.

　　[11]ShahraraS.，Huang Q.，Mandelin II A. M.，et al. TH-17 cells in rheumatoid arthritis.Arthritis Research and Therapy. 2008. 10(4): p. R93.

　　[12]KomiyamaY.，Nakae S.，Matsuki T_，et al. IL-17 plays an important role in thedevelopment of experimental autoimmune encephalomyelitis. The Journal ofImmunology. 2006. 177(1): p. 566-573.

　　[13]Bettelli E” Carrier Y?，Gao W，et al. Reciprocal developmental pathways for thegeneration of pathogenic effector TH17 and regulatory T cells. Nature. 2006.441(7090): p. 235-238.

　　[14]VeldhoenM.，Hocking R. J.，Atkins C. J.，et al. TGFp in the context of aninflammatory cytokine milieu supports de novo differentiation of IL-17-producing Tcells. Immunity. 2006. 24(2): p. 179-189.

　　[15]ManganP. R., Harrington L. E.，OQuinn D. B.，et al. Transforming growth factor-pinduces development of the TH17 lineage. Nature. 2006. 441(7090): p. 231-234.

　　[16]AkimzhanovA. M.，Yang X. 0.,Dong C. Chromatin remodeling of interleukin-17(IL-17)-IL-17F cytokine gene locus during inflammatory helper T cell differentiation.Journal of biological chemistry. 2007. 282(9): p. 5969-5972.

　　[17]NurievaR” Yang X. 0” Martinez G，et al. Essential autocrine regulation by IL-21 inthe generation of inflammatory T cells. Nature. 2007. 448(7152): p. 480-483.

　　[18]Yang L，Anderson D. E.，Baecher-Allan C.，et al. IL-21 and TGF-&bgr; are requiredfor differentiation of human TH17 cells. Nature. 2008.454(7202): p. 350-352.

　　[19]TrinchieriG，Pflanz S.,Kastelein R. A. The IL-12 family of heterodimeric cytokines:new players in the regulation of T cell responses. Immunity. 2003. 19(5): p. 641-644.

　　[20]Zhou L，Ivanov I. I，Spolski R?，et al. IL?6 programs TH-17 cell differentiation bypromoting sequential engagement of the IL-21 and IL-23 pathways. Natureimmunology. 2007. 8(9): p. 967-974.

　　[21]AggarwalS” Ghilardi N., Xie M.-H.，et al. Interleukin-23 promotes a distinct CD4 Tcell activation state characterized by the production of interleukin-17. Journal ofBiological Chemistry. 2003. 278(3): p. 1910-1914.

　　[22]Harrington L. E., Hatton R. D.，Mangan P. R.，et al. Interleukin 17—producing CD4+effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages.Nature immunology. 2005. 6(11): p. 1123-1132.

　　[23]Yang X. 0., Panopoulos A. D.，Nurieva R.，et al. STAT3 regulates cytokine-medlatedgeneration of inflammatory helper T cells. Journal of Biological Chemistry. 2007.282(13): p. 9358-9363.

　　[24]LaurenceA.，Tato C. M.，Davidson T. S?，et al. Interleukin-2 signaling via STAT5constrains T helper 17 cell generation. Immunity. 2007. 26(3): p. 371-381.

　　[25]EberlG，Marmon S.，Sunshine M.-J.，et al. An essential function for the nuclearreceptor RORyt in the generation of fetal lymphoid tissue inducer cells. Natureimmunology. 2004. 5(1): p. 64-73.

　　[26]Yang X. 0” Pappu B. P.，Nurieva R., et al. T helper 17 lineage differentiation isprogrammed by orphan nuclear receptors RORa and RORy. Immunity. 2008. 28(1): p.29-39.

　　[27]IvanovI. I.，McKenzie B. S.，Zhou L.，et al. The Orphan Nuclear Receptor RORytDirects the Differentiation Program of Proinflammatory IL-17< sup>+ T HelperCells. Cell. 2006. 126(6): p. 1121-1133.

　　[28]QingK.，Weifeng W” Fan Y?，et al. Distinct different expression of Thl7 and Th9 cellsin coxsackie virus B3-induced mice viral myocarditis. Wol J. 2011. 8: p. 267.

　　[29]Jiang N.，Liao W”Kuang X. Effects of emodin on IL-23/IL-17 inflammatory axis，Thl7 cells and viral replication in mice with viral myocarditis. Nan Fang Y Ke DaXue Xue Bao. 2014. 34(3): p. 373-378.

　　[30]Yang F.，Wu W.-F.，Yan Y.-L.，et al. Expression of il-23/thl7 pathway in a murinemodel of coxsackie virus b3-induced viral myocarditis. Virology journal. 2011. 8(1): p.301.

　　[31]FanY” Weifeng W., Yuluan Y” et al. Treatment with a neutralizing anti-murineinterleukin-17 antibody after the onset of coxsackievirus b3-induced viral myocarditisreduces myocardium inflammation. Virol J. 2011. 8(1): p.17.

　　[32]Yu M.5 Hu J” Zhu M. X.，et al. Cardiac fibroblasts recruit Thl7 cells infiltration intomyocardium by secreting CCL20 in CVB3-induced acute viral myocarditis. CellPhysiol Biochem. 2013. 32(5): p, 1437-1450.

　　[33]XieY.，Chen R.，Zhang X.，et al. The role of Thl7 cells and regulatory T cells inCoxsackievirus B3-induced myocarditis. Virology. 2011. 421(1): p. 78-84.

　　[34]YuanJ., Yu M., Lin Q.-W., et al. Thl7 cells contribute to viral replication incoxsackievirus B3-induced acute viral myocarditis. The Journal of Immunology. 2010.185(7): p. 4004-4010.

　　[35]YuanJ.，Cao A.-L., Yu M.，et al. Thl7 cells facilitate the humoral immune response inpatients with acute viral myocarditis. Journal of clinical immunology. 2010. 30(2): p.226-234.

　　[36]Yuan J” Yu M.，Lin Q.-W., et al. Neutralization of IL-17 inhibits the production ofanti-ANT autoantibodies in CVB3-induced acute viral myocarditis. Internationalimmunopharmacology. 2010.10(3): p. 272-276.

　　[37]Liu Y?，Zhu H?，Su Z.，et al. IL-17 contributes to cardiac fibrosis followingexperimental autoimmune myocarditis by a PKCbeta/Erkl/2/NF-kappaB_dependentsignaling pathway. Int Immunol. 2012. 24(10): p. 605-612.

　　[38]BaldevianoG C., Barin J. G，Talor M. V” et al. Interleukin-17A is dispensable formyocarditis but essential for the progression to dilated cardiomyopathy. Circulationresearch. 2010. 106(10): p. 1646-1655.

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